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Genetics and autoantibodies

Autoimmune diseases (ADs) are chronic conditions initiated by the loss of immunological tolerance to self-antigens. The pathogenic hypothesis comprises a complex interaction between genetic, environmental and hormonal factors that interact with an individual over time generating a dysregulation of t...

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Detalles Bibliográficos
Autores Principales: Perricone, Carlo, Agmon-Levin, Nancy, Ceccarelli, Fulvia, Valesini, Guido, Anaya, Juan-Manuel, Shoenfeld, Yehuda
Formato: Artículo (Article)
Lenguaje:Inglés (English)
Publicado: 2013
Materias:
Autoantibody
Autoimmune regulator protein
Deoxyribonuclease i
Dna methyltransferase 1
Hla dqb1 antigen
Hla drb1 antigen
Interferon regulatory factor 7
Interleukin 10
Mitochondrion antibody
Neutrophil cytoplasmic antibody
Phospholipid antibody
Rheumatoid factor
Stat4 protein
Thyrotropin receptor antibody
Transforming growth factor beta
Tumor necrosis factor receptor associated factor 1
Vitamin d
Age
Allele
Antigen presentation
Article
Autoimmune disease
Autoimmune hepatitis
Autoimmune thyroiditis
Autoimmunity
B lymphocyte
Cd8+ t lymphocyte
Dermatomyositis
Diet
Disease course
Dna polymorphism
Enteritis
Epstein barr virus
Epstein barr virus infection
Gender
Gene interaction
Genetic association
Genetic predisposition
Genetic variability
Genetics
Habit
Hla system
Hormone determination
Human
Human genome
Hypertension
Hypothesis
Immune response
Immune system
Immunogenicity
Immunoglobulin a deficiency
Immunological tolerance
Insulin dependent diabetes mellitus
Juvenile dermatomyositis
Lymphocyte differentiation
Lymphocyte proliferation
Mononucleosis
Multiple sclerosis
Nonhuman
Pathophysiology
Pemphigus
Primary biliary cirrhosis
Priority journal
Saccharomyces cerevisiae
Single nucleotide polymorphism
Sjoegren syndrome
Smoke
Systemic lupus erythematosus
Systemic sclerosis
T lymphocyte
Ulcerative colitis
Vasculitis
Vitamin blood level
Animals
Autoantibodies
Autoantigens
Autoimmune diseases
Gene-environment interaction
Genetic predisposition to disease
Hla antigens
Homeostasis
Hormones
Humans
Self tolerance
Genetic
Polymorphisms
genetic
Polymorphism
Acceso en línea:https://repository.urosario.edu.co/handle/10336/24276
https://doi.org/10.1007/s12026-013-8396-9

Internet

https://repository.urosario.edu.co/handle/10336/24276
https://doi.org/10.1007/s12026-013-8396-9

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