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Population genetics study of isoniazid resistance mutations and evolution of multidrug-resistant Mycobacterium tuberculosis

The molecular basis for isoniazid resistance in Mycobacterium tuberculosis is complex. Putative isoniazid resistance mutations have been identified in katG, ahpC, inhA, kasA, and ndh. However, small sample sizes and related potential biases in sample selection have precluded the development of stati...

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Detalles Bibliográficos
Autores Principales:	Hazbón M.H., Brimacombe M., Del Valle M.B., Cavatore M., Guerrero M.I., Varma-Basil M., Billman-Jacobe H., Lavender C., Fyfe J., García-García L., León C.I., Bose M., Chaves F., Murray M., Eisenach K.D., Sifuentes-Osornio J., Cave M.D., De León A.P., Alland D.
Formato:	Artículo (Article)
Lenguaje:	Inglés (English)
Publicado:	2006
Materias:	Dna Ethambutol Isoniazid Ahpc gene Allele Article Gene Gene identification Gene interaction Gene mutation Human Inha gene Kasa gene Katg gene Molecular genetics Multidrug resistance Mycobacterium tuberculosis Ndh gene Nonhuman Nucleotide sequence Phenotype Population genetics Priority journal Promoter region Statistical analysis Strain difference Alleles Antitubercular agents Dna mutational analysis Evolution Gene deletion Humans Microbial sensitivity tests Mutation Open reading frames Promoter regions (genetics) Rifampin Streptomycin single nucleotide antitubercular bacterial dna intergenic multidrug-resistant Antibiotics Genes Polymorphism Sequence analysis Tuberculosis
Acceso en línea:	https://repository.urosario.edu.co/handle/10336/22522 https://doi.org/10.1128/AAC.00112-06

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