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Bystander activation and autoimmunity

"The interaction over time of genetic, epigenetic and environmental factors (i.e., autoimmune ecology) increases or decreases the liability an individual would have to develop an autoimmune disease (AD) depending on the misbalance between risk and protective effects. Pathogens have been the mos...

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Detalles Bibliográficos
Autores Principales: Pacheco Y., Acosta-Ampudia, Yeny, Monsalve, Diana M., Chang C., Gershwin M.E., Anaya, Juan-Manuel
Formato: Artículo (Article)
Lenguaje:Inglés (English)
Publicado: Academic Press 2019
Materias:
Autoimmune disease
Autoimmune hepatitis
Autoimmune thyroiditis
Autoimmunity
Bacterial infection
Bacterium
Bystander effect
Cell communication
Gap junction
Graves disease
Hashimoto disease
Host
Human
Immunological tolerance
Insulin dependent diabetes mellitus
Latent period
Legionella pneumophila
Mediator
Memory t lymphocyte
Multiple sclerosis
Nonhuman
Parasitosis
Priority journal
Review
Rheumatoid arthritis
Systemic lupus erythematosus
T lymphocyte activation
Vaccination
Virus infection
Auto-reactive t cells
Autoimmune diseases
Bystander activation
Cytokines
Infection
T-cell activation
Acceso en línea:https://repository.urosario.edu.co/handle/10336/22244
https://doi.org/10.1016/j.jaut.2019.06.012

Internet

https://repository.urosario.edu.co/handle/10336/22244
https://doi.org/10.1016/j.jaut.2019.06.012

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